N-Acetyl Selank vs N-Acetyl Semax

Tuftsin-derived heptapeptide with structural modifications that resist enzymatic degradation. Modulates GABA-A receptor complex, increases enkephalin levels in limbic system, and influences serotonin metabolism.

An enhanced derivative of Selank with improved bioavailability and extended duration through N-acetylation and C-terminal amidation. Shares Selank's anxiolytic and nootropic properties with improved enzymatic resistance.

Same ACTH(4-7) core as Semax, but structural modifications resist peptidase degradation. Enhances BDNF expression, supports dopaminergic and serotonergic neurotransmission, and modulates NMDA receptor activity.

An acetylated and amidated derivative of Semax with enhanced enzymatic stability and prolonged activity. N-terminal acetylation and C-terminal amidation protect the peptide from degradation, extending its nootropic and neuroprotective effects versus base Semax.