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Peptide Profile

Angiotensin (1-7)

Ang-(1-7) · Ang 1-7 · MasR agonist · ACE2 product

Angiotensin-(1-7) [Ang-(1-7)] is an endogenous heptapeptide hormone generated primarily through cleavage of angiotensin II by ACE2, functioning as a counter-regulatory arm of the renin-angiotensin system (RAS) by binding the Mas…

Longevity & CellularInvestigationalResearch Only
Half-life
~9–10 sec (rat)
Routes
subcutaneous, intravenous

Educational research information — not medical advice. Review primary sources and consult a qualified professional before any decision.

Educational research tools — not medical advice.

Overview

What Angiotensin (1-7) is

About Angiotensin (1-7)

Binds Mas receptor (MasR), activating nitric oxide synthase and reducing oxidative stress. Opposes TGF-β and angiotensin II signaling to reduce fibrosis. Enhances insulin sensitivity and provides cardiovascular protection.

Angiotensin-(1-7) [Ang-(1-7)] is an endogenous heptapeptide hormone generated primarily through cleavage of angiotensin II by ACE2, functioning as a counter-regulatory arm of the renin-angiotensin system (RAS) by binding the Mas receptor to promote vasodilation, anti-fibrotic, anti-inflammatory, and cardioprotective effects that oppose the vasoconstrictive actions of angiotensin II. Ang-(1-7) acts through the ACE2/Mas receptor axis to reduce oxidative stress, attenuate NF-kB-mediated inflammation, and suppress TGF-beta fibrosis signaling; the ACE2/Ang-(1-7)/Mas axis has emerged as a key regulatory pathway in cardiovascular and metabolic disease, and gained renewed research attention given ACE2's role as the SARS-CoV-2 entry receptor. A Phase 1-2 randomized clinical trial of Ang-(1-7) infusion in COVID-19 ICU patients reported preliminary safety, tolerability, and dose-response data, providing the primary indexed human pharmacokinetic evidence; broader cardiovascular protective applications are supported by preclinical data but have not been established by completed Phase 3 trials. Ang-(1-7) has no FDA approval and no approved therapeutic indication in any jurisdiction; it is an endogenous peptide under active clinical investigation as a candidate for cardiovascular, metabolic, and inflammatory conditions, with emerging human safety data but an incomplete evidence base for any specific approved clinical use.

Angiotensin (1-7) Benefits & Research Areas

cardiovascular protectionanti-fibroticvasodilationinsulin sensitivity
Pharmacokinetics

Angiotensin (1-7) half-life calculator

Compound & timing

Half-lifefrom PeptideBase data
Source: "~9–10 sec (rat)" · Angiotensin (1-7) (PeptideBase)
to show amount remaining
10 sec
0%25%50%75%100%01021314252time since dose (sec)
Plasma decay curve for Angiotensin (1-7): 46% to 50% remaining at 10 sec after the dose.
range (~9–10 sec (rat))slower half-life
46%–50%remaining in plasmaat 10 sec after the dose
Half-life
9 – 10 sec
~97% cleared after
50 sec

Model: single-dose, first-order (single-compartment) estimate. Real clearance is multi-compartment and varies by individual and route.

The Research

What the evidence says about Angiotensin (1-7)

Research Evidence

based on 9 studies · 2 RCTs · most recent 2026

Regulatory Status

Availability Status
Research Only
FDA Status
Investigational

Regulatory status reflects publicly available information and may change. This is not legal or medical advice.

Research Sources

9 sources cited · 2 strong · 1 moderate · 6 weak

2 RCTs · 1 Case series · 1 Review · 5 Animals

  • Association between renin angiotensin system and cognitive outcomes over 15 years: The Look AHEAD study.

    J Alzheimers Dis · 2026

    RCTStrongPMID 42312371
  • Angiotensin-(1-7) improves oxygenation in mechanically ventilated COVID-19 patients: a randomized phase 1-2 seamless trial

    Annals of Intensive Care · 2024

    In a phase 1-2 seamless randomized trial of 107 mechanically ventilated COVID-19 patients, angiotensin-(1-7) 10 mcg/kg/day significantly increased oxygen-free days compared with controls (median 19 vs 14 days; p=0.04), supporting its role as a pulmonary protective and vasoactive agent in acute lung injury.

    RCTn=107StrongPMID 39231898
  • Perioperative Angiotensin-(1-7) for Postoperative Cognitive Vulnerability Following Coronary Artery Bypass Surgery: A Pilot Case Series.

    Res Sq · 2026

    # Summary Research found that Angiotensin-(1-7), a compound with neurovascular and anti-inflammatory properties, was feasible and well-tolerated when administered to older adults undergoing coronary artery bypass surgery, and showed preliminary signals of preserved cognitive function and reduced neuroaxonal injury markers compared to placebo. This pilot case series suggests that Angiotensin-(1-7) may warrant further investigation as a potential strategy to mitigate postoperative cognitive impairment following cardiac surgery.

    Case seriesModeratePMID 42183346
Show 6 more sources
  • The Effect of Angiotensin (1-7) on Serum Metabolomics in Obese Type 2 Diabetic Mice.

    Metabolites · 2026

    # Summary Research found that Angiotensin (1-7) intervention in obese type 2 diabetic mice reduced blood glucose levels, decreased inflammatory markers, and improved lipid profiles while restoring pancreatic β-cell function. This study demonstrated that these metabolic improvements were associated with Angiotensin (1-7)'s regulatory effects on multiple pathways, including amino acid metabolism, lipid metabolism, insulin secretion, and cellular energy metabolism processes.

    AnimalWeakPMID 42188044
  • Severe Acute Hypertension Causes Hemolysis With Release of PEP and ACE Inhibitor.

    Hypertension · 2026

    # Summary Research found that acute severe hypertension induced by angiotensin II injection causes intravascular hemolysis, which increases prolyl endopeptidase (PEP) activity and decreases angiotensin-converting enzyme (ACE) activity in plasma. This study demonstrated that these hemolysis-related enzyme changes enhance the conversion of angiotensin II to angiotensin-(1-7) while reducing angiotensin II formation, potentially influencing acute blood pressure regulation mechanisms.

    AnimalWeakPMID 42186799
  • Differential Modulation of Spinal Angiotensin-Converting Enzymes Plays a Critical Role in the Development of Trigeminal Neuropathic Pain.

    Pharmaceuticals (Basel) · 2026

    # Summary Research found that nerve injury produces an imbalance in spinal angiotensin-converting enzymes, with increased ACE1 activity and decreased ACE2 activity in pain-processing regions of the spinal cord. This study demonstrated that restoring the ACE2 pathway and increasing angiotensin-(1-7) signaling through the Mas receptor reduced neuropathic pain in an animal model of trigeminal nerve injury, suggesting that modulating these enzyme systems may represent a novel therapeutic approach for chronic pain conditions.

    AnimalWeakPMID 42198438
  • Neuroprotective effects of telmisartan in a harmaline-induced model of essential tremor: modulation of the renin-angiotensin system and inflammatory pathways.

    Front Pharmacol · 2026

    # Primary Finding Summary Research found that telmisartan exerted neuroprotective effects in an essential tremor model by increasing angiotensin (1-7) levels and MAS receptor expression in the cerebellum, which simultaneously reduced pro-inflammatory pathways and restored the balance between excitatory and inhibitory neurotransmitters. This study demonstrated that modulating the renin-angiotensin system toward its beneficial arm—characterized by elevated angiotensin (1-7) signaling—suppressed neuroinflammation and neurodegeneration associated with tremor symptoms.

    AnimalWeakPMID 42305442
  • Converting enzyme determines plasma clearance of angiotensin-(1-7)

    Hypertension · 1998

    AnimalWeakPMID 9740616
  • Neurotherapeutic roles of the protective arm of the renin-angiotensin system: from inflammation to cognitive rescue.

    Mol Biol Rep · 2026

    ReviewTheoreticalPMID 42364023

Angiotensin (1-7) Side Effects & Safety Considerations

Research-use compound with limited human data. Evidence strength varies — see the Evidence section.

No established clinical contraindications. This is an investigational compound with limited human data and has not been evaluated by the FDA for safety in this context. Consult a qualified clinician.

Consult a qualified healthcare professional before making any health decisions. This information is educational only and does not constitute medical advice.

Frequently Asked Questions — Angiotensin (1-7)

Angiotensin-(1-7) [Ang-(1-7)] is an endogenous heptapeptide hormone generated primarily through cleavage of angiotensin II by ACE2, functioning as a counter-regulatory arm of the renin-angiotensin system (RAS) by binding the Mas receptor to promote vasodilation, anti-fibrotic, anti-inflammatory, and cardioprotective effects that oppose the vasoconstrictive actions of angiotensin II. Ang-(1-7) acts through the ACE2/Mas receptor axis to reduce oxidative stress, attenuate NF-kB-mediated inflammation, and suppress TGF-beta fibrosis signaling; the ACE2/Ang-(1-7)/Mas axis has emerged as a key regulatory pathway in cardiovascular and metabolic disease, and gained renewed research attention given ACE2's role as the SARS-CoV-2 entry receptor.

cardiovascular protection, anti-fibrotic, vasodilation, insulin sensitivity, kidney protection.

Research on Angiotensin (1-7) primarily documents effects related to cardiovascular protection and anti-fibrotic and vasodilation and insulin sensitivity and kidney protection. These are areas covered in preclinical and clinical literature — individual response varies and effects depend on context of use.

For Angiotensin (1-7), its FDA status is investigational (not FDA-approved), and it is designated for research use only. Regulatory status reflects publicly available information and may change. This is not legal or medical advice.

In a phase 1-2 seamless randomized trial of 107 mechanically ventilated COVID-19 patients, angiotensin-(1-7) 10 mcg/kg/day significantly increased oxygen-free days compared with controls (median 19 vs 14 days; p=0.04), supporting its role as a pulmonary protective and vasoactive agent in acute lung injury.

Access

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