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Educational use only — not medical advice

Research library

N-Acetyl Semax benefits

From the PeptideBase research library. Educational only — not medical advice.

Studied effects

  • BDNF and NGF upregulation extrapolated from parent compound semax research
  • dopaminergic and serotonergic modulation (semax mechanism basis)
  • acetylation proposed to improve enzymatic stability vs base semax
  • no independent PubMed evidence for this specific modified compound

Mechanism

Same ACTH(4-7) core as Semax, but structural modifications resist peptidase degradation. Enhances BDNF expression, supports dopaminergic and serotonergic neurotransmission, and modulates NMDA receptor activity.

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